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Comparing transcranial magnetic stimulation and esketamine treatment response trajectories in resistant depression.

Lindsay L Benster, Jordan N Kohn, Benjamin Wade, Noah Stapper, Cory R Weissman, Jean-philippe Miron, Zafiris J Daskalakis, Lawrence G Appelbaum

Journal of affective disorders November 1, 2026 Peer reviewed DOI: 10.1016/j.jad.2026.122107 via PubMed

Summary

Esketamine was associated with a faster clinical response in adults with treatment-resistant depression compared to rTMS. Patients receiving esketamine showed a median response time of 36 days, while those on rTMS had a median response time of 49 days. Cumulative response rates were higher for esketamine at 68.8% compared to 59.4% for rTMS, though both treatments had comparable overall efficacy. Additionally, suicidal ideation improved more quickly with esketamine.

Study at a glance

Design retrospective analysis
Sample size 372
Population adults with treatment-resistant depression treated with rTMS or intranasal esketamine
Key finding Esketamine demonstrated earlier response and improvement in suicidal ideation compared to rTMS.

Abstract

Repetitive transcranial magnetic stimulation (rTMS) and intranasal esketamine are FDA-approved for treatment-resistant depression (TRD), yet comparative real-world data on response trajectories and predictors of outcomes remain limited. A retrospective analysis was performed using electronic medical records from UC San Diego Health. Adults with TRD treated with rTMS (n = 279) or intranasal esketamine (n = 93) between 2017 and 2025 were included. The primary outcome was clinical response (≥50% PHQ-9 reduction). Time-to-response was assessed using inverse probability treatment weighted (IPTW) Cox models; Kaplan-Meier curves and log-rank tests provided descriptive comparisons. Covariates included age, trauma history, anxiety comorbidity, benzodiazepine use, tobacco use history, BMI, and baseline symptom severity. Secondary outcomes included remission (PHQ-9 < 5) and suicidal ideation (SI). Esketamine demonstrated earlier response over 90 days (RMST difference = -11.94 days) and faster time-to-response in the IPTW Cox model (HR = 1.62, p = 0.005); KM estimates showed median response at 36 vs. 49 days (p = 0.0096) with convergence by ∼90 days. Cumulative response and remission rates were numerically higher for esketamine (68.8% / 45.2%) than rTMS (59.4% / 40.1%), supporting a speed-of-response difference rather than superior overall efficacy. SI improved more rapidly with esketamine (median 9 vs. 26 days; p = 0.001). In the rTMS cohort, comorbid anxiety (HR = 0.69, p = 0.039) and benzodiazepine use (HR = 0.73, p = 0.046) predicted slower response, while former tobacco use predicted faster response (HR = 1.31, p = 0.006). No significant predictors emerged for esketamine. Esketamine was associated with earlier observed antidepressant and anti-suicidal improvement than rTMS. Baseline factors, including benzodiazepine use, may help inform expectations regarding rTMS response trajectory.

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