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Elisa Hawkins

Departments of Neurology (A.R.-D., E.H., L.S.D.), Pharmacology and Toxicology (L.S.D.), School of Medicine, Virginia Commonwealth University, Richmond, Virginia and Department of Pharmacotherapy and Outcome Sciences (D.Y.A.S., F.M.J., J.L.M.), School of Pharmacy, Virginia Commonwealth University, Richmond, Virginia.

1 paper in the library · 10 citations · publishing 2024

Papers

Ketamine Produces Antidepressant Effects by Inhibiting Histone Deacetylases and Upregulating Hippocampal Brain-Derived Neurotrophic Factor Levels in a Diisopropyl Fluorophosphate-Based Rat Model of Gulf War Illness.

The Journal of pharmacology and experimental therapeutics January 17, 2024 Ana Ribeiro-Davis, Dalia Y Al Saeedy, Fay M Jahr et al. 10 citations

In a rat model of Gulf War Illness (GWI), a single antidepressant dose of ketamine reversed epigenetic changes in the hippocampus. Ketamine inhibited the upregulation of histone deacetylase enzymes, restored acetylation at the Bdnf gene promoter, and increased brain-derived neurotrophic factor (BDNF) protein expression. It also increased dendritic spine density and altered spine types, shifting from S-type to T-type spines. These results suggest ketamine's antidepressant effect in GWI-related depression involves histone modifications that boost BDNF and reshape synaptic connections, supporting further clinical trials for GWI-related depression.