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Accurate and Interpretable Prediction of Antidepressant Treatment Response from Receptor-informed Neuroimaging

Hanna M. Tolle, Andrea I Luppi, Timothy Lawn, Leor Roseman, David Nutt, Robin L. Carhart-Harris, Pedro A. M. Mediano

bioRxiv (Cold Spring Harbor Laboratory) preprint DOI: 10.1101/2025.07.02.662710

Summary

AI-generated from the abstract

A geometric deep learning model called graphTRIP predicts post-treatment depression severity from pretreatment clinical and brain imaging data. Trained on a clinical trial comparing psilocybin and escitalopram, it achieves strong predictive accuracy (r = 0.75) and generalizes to an independent dataset. The model links better outcomes to reduced functional coupling within serotonin systems and broader serotonergic integration with sensory-motor networks. Causal analysis shows a group-level advantage of psilocybin over escitalopram but identifies individuals with specific stress-related neuromodulatory profiles who may benefit more from escitalopram, advancing precision medicine and biomarker discovery in depression.

Study at a glance

Characteristics Clinical trial
Population Patients with major depressive disorder
Interventions Psilocybin Escitalopram
Topics Psychedelic-assisted therapy
Keywords Depression treatment Antidepressant therapy Mdd treatment Psilocybin therapy
Citations 1
Registration NCT03429075
Key finding The graphTRIP model accurately predicts post-treatment depression severity using pretreatment data and identifies that reduced functional coupling within serotonin systems and broader serotonergic integration with sensory-motor networks are linked to better outcomes, with psilocybin showing a group-level advantage over escitalopram but escitalopram benefiting individuals with specific stress-related neuromodulatory profiles.

Abstract

Conventional antidepressants show moderate efficacy in treating major depressive disorder. Psychedelic-assisted therapy holds promise, yet individual responses vary, underscoring the need for predictive tools to guide treatment selection. Here, we present graphTRIP (graph-based Treatment Response Interpretability and Prediction) – a geometric deep learning architecture that enables three advances: 1) accurate prediction of post-treatment depression severity using only pretreatment clinical and neuroimaging data; 2) identification of robust, patient-specific biomarkers; and 3) causal analysis of treatment effects and underlying mechanisms. Trained on data from a clinical trial comparing psilocybin and escitalopram (NCT03429075), graphTRIP achieves strong predictive accuracy (r = 0.75, p < 10−8), and generalises both to an independent dataset and across brain atlases. The model links better outcomes to reduced functional coupling within serotonin systems, and broader serotonergic integration with sensory-motor networks. Finally, causal analysis reveals a group-level advantage of psilocybin over escitalopram, but also identifies individuals with specific stress-related neuromodulatory profiles who may benefit more from escitalopram. Overall, this work advances precision medicine and biomarker discovery in depression.

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