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Ying Wei

Department of Anesthesiology, Fujian Maternity and Child Health Hospital, College of Clinical Medicine for Obstetrics & Gynecology and Pediatrics, Fujian Medical University, Fuzhou, People's Republic of China.

2 papers in the library · 15 citations · publishing 2024-2025

Papers

Intranasal Dexmedetomidine-Esketamine Combination Premedication versus Monotherapy for Reducing Emergence Delirium and Postoperative Behavioral Changes in Pediatric Tonsillectomy and/or Adenoidectomy: A Randomized Controlled Trial.

Drug design, development and therapy January 1, 2024 Yanling Liao, Siyu Xie, Yifen Zhuo et al. 14 citations

In children aged 2-5 years undergoing tonsillectomy and/or adenoidectomy with sevoflurane anesthesia, intranasal premedication combining dexmedetomidine and esketamine reduces emergence delirium more effectively than esketamine alone (9.4% vs 38.1%) and reduces postoperative negative behavioral changes at day 7 more effectively than dexmedetomidine alone (28.1% vs 48.4%). The combination also provides better sedation, easier separation from parents, better mask acceptance, shorter emergence time, and higher parental satisfaction than either drug alone, without significant adverse effects. No significant difference in emergence delirium was found between the combination and dexmedetomidine alone.

Non-targeted and Targeted Metabolomics Techniques Reveal Striatal Metabolome Characteristics in the Ketamine-Induced Conditioned Place Preference Mice Model.

Journal of molecular neuroscience : MN July 12, 2025 Weihao Fan, Yi Ye, Hongkun Yang et al. 1 citation

Ketamine, originally developed as an anesthetic, is now being studied for depression treatment, but its addictive potential is a growing concern. This research used a mouse model of ketamine-induced conditioned place preference (CPP) to investigate changes in the striatum, a brain region involved in reward. Advanced metabolomics techniques revealed that ketamine abuse alters striatal metabolites, affecting pathways related to arginine synthesis, purine metabolism, and morphine addiction. Specifically, ketamine increased the neurotransmitter kynurenine (Kyn) and decreased dopamine (DA) in the striatum. These disturbances in Kyn and DA metabolism may underlie the addictive behaviors seen in the CPP model, offering new insights into ketamine addiction mechanisms.