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Qing-Song Liu

Department of Pharmacology and Toxicology, Medical College of Wisconsin, Milwaukee, WI, 53226, USA. qsliu@mcw.edu.

2 papers in the library · 26 citations · publishing 2024

Papers

Psilocybin analog 4-OH-DiPT enhances fear extinction and GABAergic inhibition of principal neurons in the basolateral amygdala.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology April 1, 2024 Thomas J Kelly, Emma M Bonniwell, Lianwei Mu et al. 26 citations

4-OH-DiPT, a fast-acting and shorter-lasting derivative of psilocybin, reduces learned fear responses in mice by enhancing inhibitory signaling in the brain. It activates 5-HT2A receptors on interneurons in the basolateral amygdala, increasing GABAergic inhibition of principal neurons. In female mice, 4-OH-DiPT before extinction training reduced freezing to conditioned cues and later decreased avoidance behaviors in several tests, while male mice showed no significant differences. The compound acts as a near full agonist at 5-HT2A receptors and has comparable activity at mouse and human 5-HT2A/2B/2C receptors. These findings suggest a potential mechanism for suppressing learned fear.

Exploring the therapeutic potential of psychedelics: Fear extinction mechanisms and amygdala modulation

Psychedelics August 9, 2024 Thomas J. Kelly, Qing-Song Liu

Classical psychedelics are being studied as potential treatments for PTSD. Research in rodents shows these substances affect fear learning, recall, and extinction. The amygdala, a brain region central to fear processing, is key to these effects. Psychedelics interact with different cell types in the amygdala, and specific neural circuits may underlie their fear-suppressing effects. Because rodent and human amygdalas are functionally similar, findings from animal studies can guide clinical trials for psychedelic-assisted PTSD therapy. The authors emphasize that each psychedelic's unique pharmacology and duration of action are important factors for future research.