Skip to content

Journal of Nuclear Medicine

ISSN 2159-662X

3 papers in the library · 100 citations · publishing 2010-2022

Papers

Radiosynthesis and Evaluation of 11C-CIMBI-5 as a 5-HT2A Receptor Agonist Radioligand for PET

Journal of Nuclear Medicine October 18, 2010 Anders Ettrup, Mikael Palner, Nic Gillings et al. 50 citations

A radiolabeled agonist tracer, (11)C-CIMBI-5, was developed for PET imaging of the serotonin 2A (5-HT2A) receptor in the brain. In vitro assays showed CIMBI-5 is a high-affinity agonist at the 5-HT2A receptor. Ex vivo rat studies demonstrated a specific binding ratio of 0.77 ± 0.07 in the frontal cortex, which was reduced to cerebellar levels after ketanserin treatment, indicating selective binding. PET studies in pigs revealed a cortical binding potential of 0.46 ± 0.12 and a target-to-background ratio similar to the antagonist tracer (18)F-altanserin. Ketanserin treatment reduced cortical binding to cerebellar levels, confirming selective in vivo binding. (11)C-CIMBI-5 is a promising tool for investigating 5-HT2A agonist binding in the living human brain.

The Use of Microdosing in the Development of Small Organic and Protein Therapeutics

Journal of Nuclear Medicine May 25, 2017 Mats Bergström 31 citations

Microdosing uses very low, radionuclide-labeled drug doses for early human imaging or pharmacokinetic studies, requiring only a reduced preclinical toxicology package to lower cost and speed development. The approach limits exposure and risk by testing small populations for short periods. However, extrapolating results to therapeutic doses demands special attention to low-dose behavior, especially for small organic molecules versus protein therapeutics. The article reviews these aspects and discusses microdosing's role in drug development according to three pillars: tissue distribution and target access, target engagement, and induction of relevant tissue responses.

Neurovascular Uncoupling: Multimodal Imaging Delineates the Acute Effects of 3,4-Methylenedioxymethamphetamine

Journal of Nuclear Medicine September 29, 2022 Tudor M. Ionescu, Mario Amend, Tadashi Watabe et al. 19 citations

MDMA triggers neuronal activation in limbic projection areas involved in emotional processing, shown by localized increases in glucose metabolism measured with 18F-FDG fPET. Simultaneously, it causes global cerebral and extracerebral hemodynamic decreases detected by BOLD fMRI. The hemodynamic reductions strongly correlate with serotonin transporter occupancy and are of a nonneuronal, peripheral origin. Increased serotonin from SERT blockage leads to neurovascular uncoupling via direct vascular effects. These findings challenge interpretations of previous fMRI studies that suggested MDMA mainly inhibits brain activity, and recommend 18F-FDG fPET as a more robust measure for pharmacological research on psychedelics.