The action of some ergot derivatives, mescaline and dibenamine on the metabolism of separated mammalian cerebral tissues

In separated mammalian cerebral tissues, ergot derivatives, mescaline, and dibenamine each altered tissue metabolism. The substances affected the respiration and glycolysis of the tissues, with the specific effects depending on the compound and concentration used. The findings indicate that these agents can directly influence the metabolic activity of brain tissue.

Study at a glance

Abstract

Research Article| August 01 1954 The action of some ergot derivatives, mescaline and dibenamine on the metabolism of separated mammalian cerebral tissues Joyce L. Lewis; Joyce L. Lewis 1Department of Biochemistry, Institute of Psychiatry (British Postgraduate Medical Federation, University of London), Maudsley Hospital, S.E. 5 Search for other works by this author on: This Site PubMed Google Scholar H. McIlwain H. McIlwain 1Department of Biochemistry, Institute of Psychiatry (British Postgraduate Medical Federation, University of London), Maudsley Hospital, S.E. 5 Search for other works by this author on: This Site PubMed Google Scholar Biochem J (1954) 57 (4): 680–684. https://doi.org/10.1042/bj0570680 Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Facebook Twitter LinkedIn Email Cite Icon Cite Get Permissions Citation Joyce L. Lewis, H. McIlwain; The action of some ergot derivatives, mescaline and dibenamine on the metabolism of separated mammalian cerebral tissues. Biochem J 1 August 1954; 57 (4): 680–684. doi: https://doi.org/10.1042/bj0570680 Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsBiochemical Journal Search Advanced Search This content is only available as a PDF. © 1954 CAMBRIDGE UNIVERSITY PRESS1954 Article PDF first page preview Close Modal You do not currently have access to this content.