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Diana Dias-Da-Silva

IINFACTS-Institute of Research and Advanced Training in Health Sciences and Technologies, Department of Sciences, University Institute of Health Sciences (IUCS), CESPU, CRL, 4585-116 Gandra, Portugal.

2 papers in the library · 3 citations · publishing 2025

Papers

Mexican calea (Calea zacatechichi Schltdl.) interferes with cholinergic and dopaminergic pathways and causes neuroglial toxicity.

Journal of ethnopharmacology January 30, 2025 Maria Rita Garcia, Federico Ferreres, Tiago Mineiro et al. 3 citations

An aqueous extract of the aerial parts of Calea zacatechichi, a plant traditionally used for its dream-inducing effects, interferes with the cholinergic and dopaminergic systems by inhibiting acetylcholinesterase and tyrosinase, but does not affect monoamine oxidase A. The extract also shows notable cytotoxicity in neuronal and microglial cells at low concentrations, with evidence of apoptosis and necroptosis, though it scavenges free radicals and inhibits lipid peroxidation. Twenty-eight phenolic constituents were identified, 24 previously unreported in this species. The findings highlight the need for a regulatory framework for recreational use and help clarify the plant's psychopharmacological mechanisms.

Neurotoxic and Neuroprotective Effects of Psychedelics in a Human Neuroblastoma Cell Model

RevSALUS - Revista Científica da Rede Académica das Ciências da Saúde da Lusofonia January 1, 2025 Andreia Machado Brito Da Costa, Ricardo Jorge Dinis-Oliveira, Áurea Madureira-Carvalho et al.

Psychedelic compounds such as LSD, psilocin, psilocybin, 5-MeO-DMT, and mescaline show distinct neurotoxicity profiles in human neuroblastoma cells. LSD was the most cytotoxic, with EC50 values of 0.23 mM (MTT) and 0.57 mM (NR), while psilocin showed moderate toxicity (EC50 0.42 mM MTT, 0.69 mM NR). Psilocybin did not reach an EC50 within the tested range, indicating minimal toxicity. 5-MeO-DMT and mescaline affected cell viability only at higher concentrations (EC50 1.17–1.69 mM). Pre-treatment with any of these compounds did not significantly protect against glutamate-induced toxicity, suggesting limited neuroprotective potential in this model.