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Igor Horrillo

Department of Pharmacology, University of the Basque Country UPV/EHU, Leioa, Bizkaia, Spain; Centro de Investigación Biomédica en Red de Salud Mental, Instituto de Salud Carlos III, Spain; Biobizkaia Health Research Institute, Barakaldo, Bizkaia, Spain.

3 papers in the library · 134 citations · publishing 2022-2025

Papers

Serotonin 5-HT2A, 5-HT2c and 5-HT1A receptor involvement in the acute effects of psilocybin in mice. In vitro pharmacological profile and modulation of thermoregulation and head-twich response

Biomedicine & Pharmacotherapy August 29, 2022 Ines Erkizia-Santamaría, R. Alles-Pascual, Igor Horrillo et al. 108 citations

Psilocybin, a psychedelic drug that activates the 5-HT2A receptor, shows potential for treating neuropsychiatric diseases. In mice, psilocin (the active metabolite) binds with similar affinity to 5-HT2A, 5-HT2C, and 5-HT1A receptors. Psilocybin causes a dose-dependent head-twitch response, a sign of psychosis-like effects, which is blocked by a 5-HT2A antagonist but increased by a 5-HT2C antagonist. Body temperature rises at low doses but falls at higher doses; a 5-HT1A antagonist reverses this drop, causing hyperthermia. These findings clarify the roles of specific serotonin receptors in psilocybin's acute effects, aiding understanding of its therapeutic and side effects.

Evaluation of behavioural and neurochemical effects of psilocybin in mice subjected to chronic unpredictable mild stress

Translational Psychiatry June 14, 2025 Ines Erkizia-Santamaría, Igor Horrillo, Nerea Martínez-Álvarez et al. 16 citations

In a mouse model of chronic unpredictable mild stress, two doses of psilocybin (1 mg/kg, given 7 days apart) reversed stress-induced anhedonia and behavioral despair, but not apathy-related behavior. Psilocybin also produced an anxiolytic-like effect. However, it did not reverse stress-induced physiological signs of a hyperactive HPA axis or restore decreased brain-derived neurotrophic factor (BDNF) in the cerebral cortex. Psilocybin increased expression and function of serotonin-2A receptors in the cortex of both control and stressed mice, and selectively increased glucocorticoid receptor expression in the cortex of stressed mice. These findings suggest psilocybin can rescue certain depressive and anxiety-like behaviors without normalizing all stress-related physiological or neuroplasticity impairments.

Clinical and preclinical evidence of psilocybin as antidepressant. A narrative review

Progress in Neuro-Psychopharmacology and Biological Psychiatry January 1, 2025 Ines Erkizia-Santamaría, Igor Horrillo, J. Javier Meana et al. 10 citations

Psilocybin, along with its active metabolite psilocin, shows promise as a treatment for neuropsychiatric disorders. Clinical trials report that psilocybin produces a large, rapid, and lasting improvement in symptoms of depression and anxiety, with a favorable safety profile of low toxicity and good tolerance. Preclinical studies in naïve animals and animal models of disease have yielded somewhat discrepant results in standard tests for depression- and anxiety-like behavior, but overall suggest positive outcomes. This review provides an overview of clinical trials of psilocybin-assisted psychotherapy and surveys up-to-date preclinical research, including rodent models used to study the neurobiological and behavioral actions of psilocybin and its primary molecular target, the serotonin 2A receptor.