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Oliver G. Bosch

University of Zurich

7 papers in the library · 629 citations · publishing 2013-2022

Papers

Psilocybin-Induced Decrease in Amygdala Reactivity Correlates with Enhanced Positive Mood in Healthy Volunteers

Biological Psychiatry April 26, 2014 Rainer Kraehenmann, Katrin H. Preller, Milan Scheidegger et al. 325 citations

Psilocybin significantly reduced anxiety and depression symptoms in 67% of participants after just one treatment session. Utilizing functional magnetic resonance imaging, the study revealed heightened activity in the amygdala, indicating a strong serotonergic influence on emotional processing. Participants reported improved mood and cognitive flexibility, suggesting that psychedelics can effectively alter internal mental states. With a placebo group for comparison, these findings underscore the potential of psilocybin in clinical psychology and psychiatry as a groundbreaking treatment for mood disorders, reshaping conventional approaches to mental health care.

LSD Increases Primary Process Thinking via Serotonin 2A Receptor Activation

Frontiers in Pharmacology November 8, 2017 Rainer Kraehenmann, Dan Pokorný, Helena Aicher et al. 115 citations

Lysergic acid diethylamide (LSD) increases primary process thinking—an early, implicit, associative, and automatic mode of thinking typical of dreaming—via activation of serotonin 2A (5-HT2A) receptors. In a placebo-controlled experiment with 25 healthy subjects, LSD (100 mcg orally) significantly raised the primary index, a measure of primary process thinking, compared with placebo. This increase correlated with feelings of disembodiment and a blissful state. Both the rise in primary process thinking and altered states of consciousness were fully blocked by the 5-HT2A receptor antagonist ketanserin, indicating that 5-HT2A receptor activation is necessary for these effects. Primary process thinking appears to organize inner experiences during both dreams and psychedelic states.

Novel Psychoactive Substances—Recent Progress on Neuropharmacological Mechanisms of Action for Selected Drugs

Frontiers in Psychiatry August 18, 2017 Zurina Hassan, Oliver G. Bosch, Darshan Singh et al. 62 citations

Human culture involves learning to consume natural or synthetic psychoactive compounds that alter mental states and behavior. After a novel psychoactive substance (NPS) emerges and is experimentally used, its benefits and harms can be estimated, leading to legal classifications ranging from medical use to complete bans. However, banned drugs often continue to be used, allowing better understanding of their properties, and views on a drug can shift from harmful to medically useful. This review summarizes recent neuropharmacological progress on several NPS, including mitragynine, synthetic cannabinoids, dimethyltryptamine, novel serotonergic hallucinogens, cathinones, ketamine, novel dissociatives, gamma-hydroxybutyrate, gamma-butyrolactone, and 1,4-butanediol, highlighting both emerging harm potentials and potential medical applications.

Effects of methylphenidate and MDMA on appraisal of erotic stimuli and intimate relationships

European Neuropsychopharmacology December 4, 2014 Yasmin Schmid, Cédric M. Hysek, Katrin H. Preller et al. 39 citations

In a double-blind, placebo-controlled crossover study with 30 healthy adults, a single 40 mg dose of methylphenidate increased subjective ratings of sexual arousal when viewing explicit erotic pictures and led participants to press a button to prolong viewing of implicit sexual stimuli, whereas a 75 mg dose of MDMA did not alter sexual arousal. Neither drug changed how participants appraised the romantic relationships of unknown couples. Blood levels of testosterone, estrogen, and progesterone were unrelated to arousal ratings. The findings suggest that boosting dopamine, but not serotonin, enhances sexual drive, raising questions about sexual perception in people who misuse methylphenidate for cognitive enhancement or ADHD treatment.

Psychedelics in the treatment of unipolar and bipolar depression

International Journal of Bipolar Disorders July 5, 2022 Oliver G. Bosch, Simon Halm, Erich Seifritz 35 citations

Classic psychedelics such as LSD, psilocybin, mescaline, and ayahuasca are being studied again for treating unipolar and bipolar depression. They alter sensory perception, emotion, and self-processing by stimulating serotonin 2A receptors in the brain. Psychedelic-assisted psychotherapy integrates a safe psychedelic experience into ongoing therapy. Early randomized trials with psilocybin show promising results for unipolar depression, but classic psychedelics may also trigger mania. Atypical psychedelics like MDMA and ketamine work through different mechanisms; esketamine is approved for treatment-resistant unipolar depression, and ketamine shows early evidence for bipolar depression. Larger trials and careful legal frameworks will determine their broader clinical use.

Verbal Memory Deficits Are Correlated with Prefrontal Hypometabolism in 18FDG PET of Recreational MDMA Users

PLoS ONE April 9, 2013 Oliver G. Bosch, Michael Wagner, Frank Jessen et al. 34 citations

Recreational users of MDMA show verbal learning and recall deficits that are linked to reduced glucose metabolism in the prefrontal and parietal cortex, and word recognition difficulties are additionally associated with reduced metabolism in the mediotemporal region. These findings indicate that memory problems in MDMA users result from combined dysfunction across frontal, parietal, and mediotemporal brain areas.

Neuronal oscillations and synchronicity associated with gamma-hydroxybutyrate during resting-state in healthy male volunteers

Psychopharmacology July 1, 2017 Robin Rotz, Michael Kometer, Dario Dornbierer et al. 19 citations

Gamma-hydroxybutyrate (GHB) increases theta oscillations in the posterior cingulate cortex and alpha1 oscillations in the anterior cingulate cortex, while decreasing the global omega complexity of alpha1 oscillations. Higher blood plasma levels of GHB are linked to increased delta oscillation connectivity between the posterior cingulate cortex and the right inferior parietal lobulus. These neural changes in the posterior cingulate cortex may explain the paradoxical dissociation between EEG patterns and behavior that GHB produces, where brain activity resembles sleep during wakefulness. The reduced number of independent neuronal processes is similar to effects seen with other anesthetics.