Skip to content

Maurizio S Riga

Andalusian Center for Molecular Biology and Regenerative Medicine, Seville, Spain.

4 papers in the library · 186 citations · publishing 2014-2024

Papers

The natural hallucinogen 5-MeO-DMT, component of Ayahuasca, disrupts cortical function in rats: reversal by antipsychotic drugs.

The international journal of neuropsychopharmacology August 1, 2014 Maurizio S Riga, Guadalupe Soria, Raúl Tudela et al. 75 citations

5-MeO-DMT, a natural hallucinogen found in ayahuasca, disrupts brain activity in the medial prefrontal cortex (mPFC) of rodents, increasing firing in 51% and decreasing it in 35% of pyramidal neurons, while reducing the power of low-frequency cortical oscillations (<4 Hz) by 31%. This effect, which depends on 5-HT1A and 5-HT2A receptor activation, resembles disruptions caused by other psychotomimetic agents like phencyclidine and DOI. Antipsychotic drugs (haloperidol, clozapine, risperidone) and an mGlu2/3 agonist reversed the oscillation reduction. 5-MeO-DMT also decreased blood-oxygen level dependent (BOLD) responses in visual cortex and mPFC. The findings suggest these cortical alterations underlie hallucinogenic effects and may aid antipsychotic drug development.

The serotonin hallucinogen 5-MeO-DMT alters cortico-thalamic activity in freely moving mice: Regionally-selective involvement of 5-HT1A and 5-HT2A receptors.

Neuropharmacology November 1, 2018 Maurizio S Riga, Laia Lladó-Pelfort, Francesc Artigas et al. 57 citations

The hallucinogen 5-MeO-DMT alters brain oscillations more in cortical areas than in the thalamus, particularly increasing delta power in the visual cortex of mice lacking 5-HT2A receptors. It also boosts beta-band coherence between the prefrontal cortex, visual cortex, and mediodorsal thalamus. Blocking 5-HT1A receptors with WAY-100635 prevented most of these oscillatory changes in knockout mice, suggesting 5-HT1A antagonists could help treat visual hallucinations. Effects on prefrontal theta activity and cortico-thalamic coherence may relate to antidepressant properties.

The serotonergic hallucinogen 5-methoxy-N,N-dimethyltryptamine disrupts cortical activity in a regionally-selective manner via 5-HT(1A) and 5-HT(2A) receptors.

Neuropharmacology February 1, 2016 Maurizio S Riga, Analia Bortolozzi, Letizia Campa et al. 33 citations

The hallucinogen 5-MeO-DMT reduces low-frequency cortical oscillations (<4 Hz) in the prefrontal cortex, visual cortex, somatosensory cortex, and auditory cortex of anesthetized mice. In the prefrontal cortex, this reduction occurs via 5-HT(1A) receptors, as it persists in 5-HT(2A) receptor knockout mice and is blocked by a 5-HT(1A) antagonist. In sensory areas, the effect in visual cortex also involves 5-HT(1A) receptors, while other regions require 5-HT(2A) receptors. Antipsychotic drugs reverse these disruptions, supporting the model's use for developing new treatments.

Criticality supports cross-frequency cortical-thalamic information transfer during conscious states.

eLife January 5, 2024 Daniel Toker, Eli Müller, Hiroyuki Miyamoto et al. 21 citations

Bidirectional communication between the cortex and thalamus via a specific cross-frequency channel is linked to conscious states. In humans, mice, and rats, low-frequency waves (1–13 Hz) sent from either the cortex or thalamus are consistently encoded by the other region using high gamma waves (52–104 Hz). This cross-frequency communication is diminished during propofol-induced unconsciousness and generalized spike-and-wave seizures, but enhanced by the psychedelic 5-MeO-DMT. Numerical simulations and neural recordings suggest these changes are mediated by shifts in thalamocortical electrodynamics toward or away from edge-of-chaos criticality, offering a mathematical framework for disrupted information transfer during unconsciousness.