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Irene Acero-Pousa

Universitat Pompeu Fabra

2 papers in the library · 1 citation · publishing 2026

Papers

Distinct brain responses to psilocybin and escitalopram in depression captured by the Fluctuation-Dissipation Theorem

bioRxiv (Cold Spring Harbor Laboratory) June 16, 2026 Paulina Clara Dagnino, Irene Acero-Pousa, Gorka Zamora‐lópez et al. 1 citation

Psilocybin and the conventional antidepressant escitalopram produce opposite changes in the brain's hierarchical non-equilibrium dynamics when treating major depressive disorder. Using resting-state fMRI before and after treatment, researchers built whole-brain models and measured how much each patient's brain activity deviated from the fluctuation-dissipation theorem. Baseline measures distinguished treatment responders from non-responders within each group. The deviation from the fluctuation-dissipation theorem may serve as a marker to differentiate the brain effects of psilocybin and escitalopram, contributing to understanding how these treatments work for depression.

Divergent changes in perturbation-induced brain reconfiguration following depression treatment with psilocybin and escitalopram

bioRxiv (Cold Spring Harbor Laboratory) June 26, 2026 Paulina Clara Dagnino, Irene Acero-Pousa, Robin Carhart‐Harris et al.

A central challenge in neuroscience is understanding how the human brain is organised to support optimal functioning and adaptability. One approach to characterise complex brain dynamics is by artificially perturbing whole-brain models. Here, we asked whether whole-brain organisation under perturbation in major depressive disorder (MDD) changes after intervention with psilocybin and escitalopram. First, we built whole-brain models of pre- and post-treatment resting-state functional magnetic resonance imaging (fMRI) and obtained an initial generative effective connectivity (GEC) matrix for each individual.