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Neurosci Biobehav Rev

ISSN 0149-7634; 1873-7528;

3 papers in the library · 211 citations · publishing 2022-2026

Papers

Resting-state fMRI functional connectivity and mindfulness in clinical and non-clinical contexts: A review and synthesis.

Neurosci Biobehav Rev February 22, 2022 206 citations

This review synthesizes evidence on the relationship between resting-state fMRI functional connectivity and mindfulness in both clinical and non-clinical populations. The authors find that mindfulness practice is associated with altered functional connectivity in brain networks involved in attention, self-referential processing, and emotion regulation. These changes may underlie the psychological benefits of mindfulness, but the review notes that the current evidence is mixed and limited by heterogeneous methodologies and small sample sizes.

The clinical neuroscience of lucid dreaming.

Neurosci Biobehav Rev January 14, 2025 5 citations

Lucid dreaming, where the dreamer becomes aware of being in a dream and often gains control over it, can occur spontaneously or be induced by behavioral, cognitive, or technological methods. This review examines evidence linking lucid dreaming to conditions such as nightmare disorder, depression, anxiety, psychosis, and dissociative states, and discusses possible neurobiological bases for these associations. It also explores contemplative sleep practices like Dream/Sleep Yoga and Yoga Nidrâ that train lucid states during sleep. Potential drawbacks of lucid dreaming interventions are outlined, along with impacts on individuals without clinical conditions, contributing to understanding its therapeutic possibilities and implications.

From psychache to neuroplasticity: A unified rescue-repair model of Ketamine's anti-suicidal action.

Neurosci Biobehav Rev June 7, 2026

Ketamine's rapid anti-suicidal effects may stem from a unified mechanism that addresses both psychological pain (psychache) and neuroplasticity. The proposed rescue-repair model suggests ketamine first alleviates intense emotional distress by modulating brain circuits involved in mood and cognition, then promotes neural repair and connectivity changes that sustain symptom relief. This framework integrates psychological and neurobiological perspectives, offering a more complete explanation for how a single drug can quickly reduce suicidal thoughts while also fostering long-term brain health.