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The World Journal of Biological Psychiatry

ISSN 1562-2975

7 papers in the library · 322 citations · publishing 2004-2023

Papers

Practical recommendations for the management of treatment-resistant depression with esketamine nasal spray therapy: Basic science, evidence-based knowledge and expert guidance

The World Journal of Biological Psychiatry November 3, 2020 Siegfried Kasper, Wiesław Jerzy Cubała, Andrea Fagiolini et al. 84 citations

Esketamine nasal spray, an NMDA glutamate receptor antagonist, offers a fast-acting treatment option for patients with treatment-resistant depression (TRD) who have not responded to several prior therapies. A group of six European experts with clinical experience using esketamine nasal spray developed consensus statements on practical considerations before, during, and after administration. The guidance is based on their experience and available literature, aiming to help clinicians unfamiliar with the treatment. Further real-world use is expected to expand existing knowledge.

Repeated intravenous ketamine therapy in a patient with treatment-resistant major depression

The World Journal of Biological Psychiatry May 12, 2008 Michael Liebrenz, Rudolf Stohler, Alain Borgeat 73 citations

In a 55-year-old man with treatment-resistant major depression and co-occurring alcohol and benzodiazepine dependence, a single intravenous infusion of ketamine (0.5 mg/kg) produced a pronounced improvement in depression symptoms, peaking two days later (Hamilton Depression Rating Scale down 56.6%, Beck Depression Inventory down 65.4%). Positive effects began fading by day 7 and returned to baseline by day 35. A second infusion six weeks later was less effective, reducing symptoms by 43% and 35% respectively, with effects lasting only 7 days. Repeated ketamine administration can be beneficial, but the diminished response to the second dose suggests that optimal dosing and scheduling require further investigation.

Potential antipsychotic properties of central cannabinoid (CB1) receptor antagonists

The World Journal of Biological Psychiatry March 1, 2010 Patrik Roser, Franz X. Vollenweider, Wolfram Kawohl 63 citations

Delta(9)-Tetrahydrocannabinol (THC), the main psychoactive component of cannabis, can cause psychomotor effects, psychotic reactions, and cognitive impairment similar to schizophrenia. These effects can be reduced by two other cannabinoids: cannabidiol (CBD) and SR141716. CBD, the second most abundant cannabis constituent, weakly antagonizes the CB(1) receptor, inhibits anandamide reuptake and hydrolysis, and has neuroprotective antioxidant activity. SR141716 is a potent and selective CB(1) receptor antagonist. Both can reverse many effects of CB(1) receptor agonists, suggesting antipsychotic properties. Experimental studies in animals, healthy volunteers, and schizophrenic patients support this, with a pharmacological profile similar to atypical antipsychotic drugs. This review presents preclinical and clinical studies on the potential antipsychotic effects of CBD and SR141716.

Khat and mushrooms associated with psychosis

The World Journal of Biological Psychiatry January 1, 2004 Roman J Nielen, Frank Mma van der Heijden, S. Tuinier et al. 42 citations

Two cases of khat-induced psychosis and two of psilocybin-induced psychosis are described. Khat-induced psychotic symptoms resolved without treatment within one week. One psilocybin-induced psychosis patient was treated with risperidone; the other's symptoms subsided in a few days. No somatic medical complications occurred. The paper stresses adequate psychiatric diagnosis and treatment, negative social consequences of drug use, and the need to distinguish these from functional psychoses, especially in patients with psychiatric comorbidity.

Strategies to mitigate dissociative and psychotomimetic effects of ketamine in the treatment of major depressive episodes: a narrative review

The World Journal of Biological Psychiatry January 11, 2016 Matthew Cooper, Joshua D. Rosenblat, Danielle S. Cha et al. 40 citations

Ketamine produces rapid antidepressant effects but can cause psychotomimetic and dissociative side effects, raising safety concerns. This narrative review synthesizes strategies to reduce those effects, including altering dose and infusion rate, changing the route of administration, choosing a specific enantiomer, co-administering mood stabilizers or antipsychotics, and using alternative NMDA-modulating agents like lanicemine and GLYX-13. Intranasal administration appears the most promising approach for mitigating dissociative and psychotomimetic effects, but the available studies are limited in number and quality, so further investigation is needed.

Increase in thalamic cerebral blood flow is associated with antidepressant effects of ketamine in major depressive disorder

The World Journal of Biological Psychiatry January 5, 2022 Matti Gärtner, Mischa de Rover, Lena Václavů et al. 11 citations

In patients with Major Depressive Disorder, increased blood flow in the thalamus one day after a single low-dose ketamine infusion is linked to greater improvement in depressive symptoms. Lower thalamus blood flow before treatment predicts both a larger increase in flow after ketamine and stronger symptom reduction. The study used arterial spin labelling, a brain imaging technique that directly measures cerebral blood flow, avoiding the ambiguity of standard BOLD imaging. These findings suggest that measuring regional blood flow could help guide ketamine treatment decisions.

Short term ketamine treatment in patient with bipolar disorder with comorbidity with borderline personality disorder: Focus on impulsivity

The World Journal of Biological Psychiatry June 20, 2023 Maria Gałuszko‐węgielnik, Katarzyna Jakuszkowiak‐wojten, Alina Wilkowska et al. 9 citations

A woman with both bipolar disorder and borderline personality disorder received intravenous ketamine for acute depression. While ketamine initially improved her depressed mood, continued treatment led to increased nonsuicidal self-injury, impulsive behavior, and worsening dissociative symptoms. Treatment was stopped and replaced with a different medication that proved helpful. The authors conclude that ketamine's effects on emotional dysregulation and impulsivity are unclear and differ from its antidepressant effects, highlighting the need for more safety and effectiveness studies in this patient group.