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Mitul A Mehta

Department of Neuroimaging, Institute of Psychiatry, Psychology and Neuroscience, King's College London, London, UK.

9 papers in the library · 236 citations · publishing 2013-2026

Papers

Ketamine induces a robust whole-brain connectivity pattern that can be differentially modulated by drugs of different mechanism and clinical profile

Psychopharmacology May 19, 2015 R. Joules, O. Doyle, A. J. Schwarz et al. 77 citations

Ketamine, which blocks N-methyl-D-aspartate receptors (NMDARs), robustly alters functional connectivity in the human brain, shifting patterns from cortex-centered to subcortex-centered connections. This effect was detected with 87.5% accuracy compared to saline. Pre-treatment with risperidone strongly modulated the connectivity changes (81.25% accuracy), whereas lamotrigine did not (43.75% accuracy). The differential modulation suggests the connectivity effects stem primarily from NMDAR blockade rather than downstream glutamate release. No such differential effect was seen in measures of brain response amplitude, underscoring the value of connectivity analysis for understanding how drugs affect the brain.

Modelling psychiatric and cultural possession phenomena with suggestion and fMRI.

Cortex; a journal devoted to the study of the nervous system and behavior April 1, 2014 Quinton Deeley, David A Oakley, Eamonn Walsh et al. 75 citations

Involuntary movements in neuropsychiatric disorders and culturally influenced dissociative states, such as delusions of alien control and spirit possession, involve distinct brain processes. Using fMRI in 15 highly hypnotically susceptible volunteers, suggestions modelled different experiences of loss of self-control: external personal control (like delusions of control), internal personal control (like spirit possession), and impersonal control by a machine (technical delusions). Brain activity and connectivity varied across these conditions.

Using hypnotic suggestion to model loss of control and awareness of movements: an exploratory FMRI study.

PloS one January 1, 2013 Quinton Deeley, Eamonn Walsh, David A Oakley et al. 48 citations

Voluntary control and awareness of movement are central to selfhood and responsibility, yet can be lost in neuropsychiatric syndromes and dissociative states like spirit possession. Using suggestion and fMRI in 15 highly hypnotically suggestible subjects, loss of perceived control of movements was linked to reduced connectivity between the supplementary motor area (SMA) and motor regions. Reduced awareness of involuntary movements corresponded with less activation in parietal cortices and insula. These results suggest the sense of voluntary control may critically depend on SMA coupling with motor systems, offering a neural basis for narrowed awareness in pathological and culturally influenced dissociative phenomena.

Effect of naltrexone pretreatment on ketamine-induced glutamatergic activity and symptoms of depression: a randomized crossover study.

Nature medicine July 24, 2025 Luke A Jelen, David J Lythgoe, James M Stone et al. 29 citations

Blocking the opioid system with naltrexone reduced both the brain glutamate response and the antidepressant effect of ketamine in adults with major depressive disorder. In a double-blind crossover study of 26 adults, naltrexone before ketamine infusion attenuated the rise in glutamate+glutamine relative to N-acetylaspartate in the anterior cingulate cortex and lessened the drop in depression scores the next day. The opioid system modulates ketamine's acute brain effects and subsequent mood improvement, suggesting interactions between glutamate and opioid systems may inform new depression treatments.

A randomized, double-blind, placebo-controlled, Phase 1 study to investigate the safety, tolerability, pharmacokinetics and pharmacodynamics of an immediate-release oral ketamine capsule in healthy volunteers.

Journal of psychopharmacology (Oxford, England) June 20, 2025 Mutahira Qureshi, Daniel Silman, Romayne Gadelrab et al. 3 citations

A single dose of immediate-release oral ketamine (40–240 mg) was safe and generally well-tolerated in healthy adults, with no unexpected safety signals or discontinuations due to side effects. Eighty mild or moderate treatment-emergent adverse events occurred after ketamine doses, most commonly dissociation, dizziness, and headache, while only five occurred after placebo. Dissociation events increased with higher doses. Ketamine and its metabolites showed dose-proportional pharmacokinetics. Transient mood and dissociation changes appeared one hour after dosing and resolved within about four hours. These results support further investigation of oral ketamine capsules for treatment-resistant depression.

How to set up a psychedelic study: Unique considerations for research involving human participants.

Neuroscience and biobehavioral reviews July 1, 2026 Marcus J Glennon, Catherine I V Bird, Prateek Yadav et al. 2 citations

Setting up a psychedelic research study involves a long, arduous, and Kafkaesque process with many unstandardised challenges. These complexities challenge existing assumptions about psychiatric prescribing, the placebo effect, and definitions of selfhood. This review brings together major UK psychedelic research teams to formalise these unique considerations, addressing sociocultural, political, legal, pharmacological, safety, study design, and experiential facets. It identifies continuing areas of debate and provides a practical, experience-based guide with recommendations for policymakers and future researchers intending to set up a psychedelic study or clinical trial.

Regional Blood Flow Signatures of Opioidergic Modulation of Ketamine in Major Depressive Disorder: A Randomized Crossover Study.

The American journal of psychiatry June 1, 2026 Luke A Jelen, Owen O'Daly, Fernando O Zelaya et al. 2 citations

Ketamine increased blood flow in specific brain regions (subgenual, pregenual, and dorsal anterior cingulate cortices) in adults with major depressive disorder, and this effect was not blocked by the opioid blocker naltrexone. However, naltrexone did disrupt the relationships between blood flow changes and both acute subjective effects and antidepressant response. The blood flow changes aligned with patterns of opioid and glutamate receptor distribution, suggesting that ketamine's effects involve interactions among multiple neurotransmitter systems.

Spatial collinearity constrains multivariate molecular-enriched network estimation.

bioRxiv : the preprint server for biology June 12, 2026 Timothy Lawn, Johan Nakuci, Steve Cr Williams et al.

Spatial overlap among brain receptor maps derived from PET imaging can distort analyses that model multiple receptors together. Using test-retest fMRI data, the authors show that as more receptors are included in a multivariate model, the reliability of the resulting functional connectivity networks decreases, and this degradation is driven by collinearity among the receptor maps. A univariate approach, modeling each receptor independently, produces more reliable networks and, in a study comparing LSD to placebo, better captured the known role of the 5HT-2A receptor. Spatial collinearity is a fundamental constraint on multivariate molecular-enriched network estimation, and univariate modeling is recommended as a more robust default.

Structural imaging predictors of ketamine response in treatment-resistant depression: a machine learning approach.

Translational psychiatry May 12, 2026 Linda Bryant, Laith Alexander, Sergio Mena et al.

A machine-learning model using structural brain scans predicted which adults with treatment-resistant depression would respond to a single ketamine infusion. The model, trained on 99 participants, achieved 72% balanced accuracy in the discovery sample and 60% in two independent groups, with performance dropping to chance in a saline-treated control group. Greater gray matter volume in frontal regions predicted response, while greater cerebellar volume predicted non-response. The findings suggest that pre-treatment brain structure may help guide personalized treatment decisions for ketamine therapy.