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James M Stone

Brighton and Sussex Medical School, University of Sussex, Brighton, UK.

4 papers in the library · 164 citations · publishing 2015-2026

Papers

Drug models of schizophrenia.

Therapeutic advances in psychopharmacology February 1, 2015 Hannah Steeds, Robin L Carhart-Harris, James M Stone 132 citations

Schizophrenia involves positive, negative, and cognitive symptoms, and about one-third of patients do not respond to existing medications. This review evaluates how drugs acting on dopaminergic, glutamatergic, serotonergic, cannabinoid, GABA, cholinergic, and kappa opioid systems model aspects of schizophrenia in animals and humans. Understanding interactions between these neurotransmitter systems and their links to symptoms is crucial for forming a coherent hypothesis of schizophrenia's pathogenesis and developing new therapies.

Effect of naltrexone pretreatment on ketamine-induced glutamatergic activity and symptoms of depression: a randomized crossover study.

Nature medicine July 24, 2025 Luke A Jelen, David J Lythgoe, James M Stone et al. 29 citations

Blocking the opioid system with naltrexone reduced both the brain glutamate response and the antidepressant effect of ketamine in adults with major depressive disorder. In a double-blind crossover study of 26 adults, naltrexone before ketamine infusion attenuated the rise in glutamate+glutamine relative to N-acetylaspartate in the anterior cingulate cortex and lessened the drop in depression scores the next day. The opioid system modulates ketamine's acute brain effects and subsequent mood improvement, suggesting interactions between glutamate and opioid systems may inform new depression treatments.

Regional Blood Flow Signatures of Opioidergic Modulation of Ketamine in Major Depressive Disorder: A Randomized Crossover Study.

The American journal of psychiatry June 1, 2026 Luke A Jelen, Owen O'Daly, Fernando O Zelaya et al. 2 citations

Ketamine increased blood flow in specific brain regions (subgenual, pregenual, and dorsal anterior cingulate cortices) in adults with major depressive disorder, and this effect was not blocked by the opioid blocker naltrexone. However, naltrexone did disrupt the relationships between blood flow changes and both acute subjective effects and antidepressant response. The blood flow changes aligned with patterns of opioid and glutamate receptor distribution, suggesting that ketamine's effects involve interactions among multiple neurotransmitter systems.

Association of Hallucinogen Persisting Perception Disorder with Trait Neuroticism and Mental Health Symptoms.

Journal of psychoactive drugs January 1, 2025 Morgan Hadley, Alicia Halliday, James M Stone 1 citation

Hallucinogen Persisting Perception Disorder (HPPD) may be more common than previously thought. Among 415 hallucinogen and other drug users who completed an online questionnaire, 39.7% reported symptoms of Type I HPPD and 4.3% reported symptoms of Type II HPPD. Neuroticism scores did not differ between those with and without HPPD. Individuals with Type II HPPD were more likely to report anxiety, obsessional thoughts, paranoia, hypochondria, and panic attacks, and were also more likely to have used 25I-NBOMe, dextromethorphan, nitrous oxide, and benzodiazepines. Nearly half (47.3%) had never tested their drugs, complicating attribution of HPPD severity to specific substances.