Skip to content

Patrícia Bado

Graduate Program in Psychiatry and Behavioral Sciences, Hospital de Clínicas de Porto Alegre (HCPA), Universidade Federal do Rio Grande do Sul (UFRGS), Brazil.

2 papers in the library · 50 citations · publishing 2013-2025

Papers

Functional dissociation of ventral frontal and dorsomedial default mode network components during resting state and emotional autobiographical recall

Human Brain Mapping November 6, 2013 Patrícia Bado, Annerose Engel, Ricardo de Oliveira‐souza et al. 49 citations

Mind-wandering, which occupies much of daily life, often involves autobiographical recall and self-reflection. Brain imaging shows that a set of regions called the default mode network (DMN) is active during such spontaneous thought, but the roles of different cognitive components within the DMN were unclear. Using fMRI, researchers compared brain activity during emotional autobiographical memory recall, neutral memory recall, and resting wakefulness, with a subtraction task as a control. Both emotional recall and resting state activated shared DMN regions compared to the control.

Neural basis of GAD improvement following a mindfulness-based intervention and antidepressant treatment: an analysis from a randomized controlled trial.

Psychiatry research July 12, 2025 Marianna de Abreu Costa, Patrícia Bado, Maiko Schneider et al. 1 citation

Symptom improvement in generalized anxiety disorder may involve enhanced functional coupling between brain regions critical for emotional regulation, self-referential processing, and stimulus selection, particularly between the left amygdala and right orbitofrontal cortex. In a randomized trial with 20 patients, those receiving mindfulness training showed improvement linked to amygdala connectivity with the default mode and salience networks, while those on fluoxetine showed improvement linked to amygdala-orbitofrontal cortex coupling. However, no significant differences between the two treatment groups were found. Larger studies are needed to clarify treatment-specific neural mechanisms.