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H. Valerie Curran

University College London

5 papers in the library · 1,023 citations · publishing 2000-2022

Papers

Ketamine use: a review

Addiction July 21, 2011 Celia J. A. Morgan, H. Valerie Curran 645 citations

Repeated misuse of ketamine, both acute and chronic, causes significant physical, psychological, and social harms. A major physical harm is ketamine-induced ulcerative cystitis, particularly linked to chronic, frequent use. Frequent daily use is also associated with neurocognitive impairment, most robustly deficits in working and episodic memory. Recent studies suggest neurological abnormalities that may underlie these cognitive effects. Many frequent users report trying but failing to stop using ketamine, indicating addiction concerns. Treatment for ketamine-induced ulcerative cystitis should coordinate urologists and addiction specialists. Neurocognitive impairment can negatively impact educational and work achievement and compound addiction. Prevention and harm minimization campaigns are needed to alert young people to these potentially chronic effects.

Adjunctive Ketamine With Relapse Prevention–Based Psychological Therapy in the Treatment of Alcohol Use Disorder

American Journal of Psychiatry January 11, 2022 Meryem Grabski, Amy Mcandrew, Will Lawn et al. 169 citations

Three weekly infusions of ketamine (0.8 mg/kg) helped people with severe alcohol use disorder stay abstinent more days over six months than placebo infusions did. The ketamine group averaged 10.1% more days abstinent than the placebo group. Combining ketamine with mindfulness-based relapse prevention therapy produced the largest improvement, with 15.9% more abstinent days compared with placebo plus alcohol education. No serious adverse events occurred. Relapse rates did not differ significantly between ketamine and placebo groups. The findings suggest ketamine is safe and may support abstinence, especially when paired with psychological therapy.

Is MDMA (‘Ecstasy’) Neurotoxic in Humans? An Overview of Evidence and of Methodological Problems in Research

Neuropsychobiology January 1, 2000 H. Valerie Curran 143 citations

MDMA is neurotoxic in animals, and evidence for human neurotoxicity comes from three indirect research areas: neurobiological and neuroendocrine studies, psychological and somatic symptoms in users, and psychiatric case reports. The strength of causative links between observed effects, MDMA use, and human neurotoxicity varies across these study types. Methodological problems in human MDMA research complicate interpretation.

Recreational 3,4-methylenedioxy-N-methylamphetamine (MDMA) or ‘ecstasy’ and self-focused compassion: Preliminary steps in the development of a therapeutic psychopharmacology of contemplative practices

Journal of Psychopharmacology May 18, 2015 Sunjeev K. Kamboj, Emma J. Kilford, Stephanie Minchin et al. 43 citations

MDMA (ecstasy) and compassionate imagery both increase self-compassion and reduce self-criticism in recreational users. In a non-blind experiment, participants who consumed ecstasy showed similar pro-social effects to those produced by a contemplative compassion exercise, particularly in those with higher attachment-related avoidance. The findings suggest MDMA may enhance psychotherapy by fostering compassionate attitudes toward oneself. However, because the study was not blinded and drug purity was unknown, controlled trials with pharmaceutical-grade MDMA are needed to confirm these effects.

The acute effects of cannabidiol on the neural correlates of reward anticipation and feedback in healthy volunteers

Journal of Psychopharmacology August 5, 2020 Will Lawn, J. P. Hill, Chandni Hindocha et al. 23 citations

A single 600 mg oral dose of cannabidiol did not alter brain activity related to anticipating or receiving rewards in healthy adults. Using functional magnetic resonance imaging during a monetary incentive delay task, the expected reward-related brain regions—including the insula, caudate, nucleus accumbens, anterior cingulate, and orbitofrontal cortex—were activated, but no difference was observed between cannabidiol and placebo. Bayesian analyses confirmed that activity in these regions was similar under both conditions, and behavioral measures of motivation for reward also showed no significant difference. The findings suggest that acute cannabidiol does not affect the neural correlates of reward anticipation or feedback in healthy individuals.